Lamin A/C expression in hematopoietic cells regulates mouse atherosclerosis: a potential mechanism contributing to age-dependent atherosclerosis
Smooth muscle cell-specific deletion of S100A4 reroutes smooth muscle cell fate and modifies the inflammatory status of murine atherosclerotic lesions
The epigenetic enzyme DOT1L orchestrates VSMC–monocyte crosstalk to protect against atherosclerosis via the NF-KB pathway